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Please use this identifier to cite or link to this item: http://hdl.handle.net/1959.14/116753

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Title
Snoring-related energy transmission to the carotid artery in rabbits
Related
Journal of applied physiology, Vol. 100, Issue 5 (2006), p.1547-1553
DOI
10.1152/japplphysiol.01439.2005
Publisher
American Physiological Society
Date
2006
FoR/RFCD Code(s)
110200 Cardiovascular Medicine and Haematology
Author/Creator
Amatoury, Jason
Author/Creator
Howitt, Lauren
Author/Creator
Wheatley, John R
Author/Creator
Avolio, Alberto P
Author/Creator
Amis, Terence C
Description
Epidemiological studies link habitual snoring and stroke, but mechanisms involved are poorly understood. One previously advanced hypothesis is that transmitted snoring vibration energy may promote carotid atheromatous plaque formation or rupture. To test whether vibration energy is present in carotid artery walls during snoring we developed an animal model in which we examined induced snoring (IS)-associated tissue energy levels. In six male, supine, anesthetized, spontaneously breathing New Zealand White rabbits, we surgically inserted pressure transducer-tipped catheters (Millar) to monitor tissue pressure at the carotid artery bifurcation (PCT) and within the carotid sinus lumen (PCS; artery ligated). Snoring was induced via external compression (sandbag) over the pharyngeal region. Data were analyzed using power spectral analysis for frequency bands above and below 50 Hz. For frequencies below 50 Hz, PCT energy was 2.2 (1.1–12.3) cmH₂O² [median (interquartile range)] during tidal breathing (TB) increasing to 39.0 (2.5–95.0) cmH₂O² during IS (P = 0.05, Wilcoxon's signed-rank test). For frequencies >50 Hz, PCT energy increased from 9.2 (8.3–10.4) x 10⁻⁴ cmH₂O² during TB to 172.0 (118.0–569.0) x 10⁻⁴ cmH₂O² during IS (P = 0.03). Concurrently, PCS energy was 13.4 (8.5–18.0) x 10⁻⁴ cmH₂O² during TB and 151.0 (78.2–278.8) x 10⁻⁴ cmH₂O² during IS (P < 0.03). The PCS energy was greater than PCT energy for the 100–275 Hz bandwidth. In conclusion, during IS there is increased energy around and within the carotid artery, including lower frequency amplification for PCS. These findings may have implications for carotid atherogenesis and/or plaque rupture.
Description
7 page(s)
Subject Keyword
110200 Cardiovascular Medicine and Haematology
Subject Keyword
stroke
Subject Keyword
atherosclerosis
Resource Type
journal article
Organisation
Macquarie University. Australian School of Advanced Medicine

Identifier
http://hdl.handle.net/1959.14/116753
Identifier
ISSN:8750-7587
Identifier
mq-rm-2007009790
Language
eng
Reviewed
Reviewed
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Subject
"Journal of applied physiology"
 
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